Διαβητική αμφιβληστροειδοπάθεια
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Transcript of Διαβητική αμφιβληστροειδοπάθεια
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1968 Siperstein=H 1971 Williamson= 1990 . 1990
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1
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KROCSTENOOSLOSTOCKHOLM7232461008244160MELETH
ATOMA
()
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DCCT
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1422 (99%) DCCT
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N 70-120 mg/dl
M 65 mg/dl
b1c
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ETH ME
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b1c
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27%60%76%>1>3 > 3
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N=332N=36352 (2,4)62 (2,9)49 (2,2)26 (1,1)45 (2,0)21 (1,9) Laser :/100
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34%54%22%46%54%E >3 >3 .Laser
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27 %60 %76 %34 %54 %46 %54 % >3 >3 .Laser 1 >3 >3
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HBA1c
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2
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UKPDS
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HbA1c cross-sectional, median values
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Hypoglycaemic episodes per annumActual Therapy analysis
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Glucose Control Study SummaryThe intensive glucose control policy maintained a lower HbA1c by mean 0.9 % over a median follow up of 10 years from diagnosis of type 2 diabetes with reduction in risk of:12%for any diabetes related endpointp=0.02925%for microvascular endpointsp=0.009916%for myocardial infarctionp=0.05224%for cataract extractionp=0.04621%for retinopathy at twelve yearsp=0.01533%for albuminuria at twelve yearsp=0.000054
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1148 2 A< 144 / 82 mmHg any diabetes-related endpoint24% p=0.0046diabetes-related deaths32%p=0.019stroke44%p=0.013microvascular disease37%p=0.0092 heart failure56%p=0.0043retinopathy progression34%p=0.0038deterioration of vision47%p=0.0036
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1c (~1%) /
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10
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