Fisiopatologc3ada de-tuberculosis
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TUBERCULOSIS
Dr. Pedro G. Cabrera J.
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Tuberculosis en EEUU
NEJM, Vol 345 N 3 July 2001
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Tuberculosis en Perú
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Fuente: DGSP – DEGS – DEAIS – ESNC de la Tuberculosis . Ministerio de Salud.
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TuberculosisMycobacterium tuberculosis, bacilo ácido-alcohol resistente, aerobio estricto, no espurulado, inmóvil, no productor de toxinas.
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PatogeniaDiseminación por partículas de aerosol, con lo que por medio de la respiración el germen llega al espacio alveolar.Ingeridos por macrófagos. Se produce infección cuando las bacterias inoculadas escapan.Se produce diseminación Posterior diseminación mediante vasos linfáticos hacia ganglios linfáticos regionales “complejo primario de Ghon” (neumonitis + linfagitis + adenitis)Vía hematógena alcanza el resto de órganos, donde anida en estado latente sin replicación.
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Patogenia
“Los microorganismos latentes pueden reactivarse en casos en los que se produce disminución de las defensas inmunológicas.”
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Patogenia
La mayoría de casos de TBC clínica ocurre meses o años después de la primoinfección
El principal sistema defensivo es la inmunidad celular.
Reactividad cutánea de tuberculina. Inyección intradérmica de PPD. Lectura a las 48 – 72 horas. Sólo traduce inmunidad!
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Clínica TBC
Tuberculosis respiratoria
Neumonía tuberculosa
Pleuritis tuberculosa
Tuberculosis miliar o diseminada
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Clínica TBC
Tuberculosis extrapulmonar
Meningitis tuberculosa
Tuberculosis genitourinaria
Osteomielitis tuberculosa
Adenitis tuberculosa
Serositis, pericarditis, peritonitis
Tuberculosis cutánea
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PPD (Derivado Protéico Purificado)
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PPD (Derivado Protéico Purificado)
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PPD (Derivado Protéico Purificado)
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Diagnóstico
Se basa en la demostración de la micobacteria: tinción Ziehl-Neelsen o auramina, cultivos (Lowenstein-Jensen), anticuerpos por ELISA, técnicas de PCR.
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Tratamiento
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Tratamiento
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